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Mother Nature is having a good laugh over this GlucoLife Formula one. "Let’s create a condition where humans don’t produce any insulin - necessary for making use of GlucoLife glucose formula - and then make those same humans produce too much glucagon - something that increases glucose levels. You’ve likely heard of beta-cells - which are produced by the pancreas and directly responsible for secreting the hormone insulin. In a person with type 1 diabetes, your immune system is constantly attacking and destroying your body’s beta-cells. You make ‘em, you destroy ‘em. Glucagon, on the other hand, is a hormone secreted by alpha-cells, also produced by your pancreas. This hormone tells your liver to release glycogen - which is basically stored sugar. Your liver then releases glycogen, it’s converted into glucose, enters your bloodstream, and raises your blood sugar. In a person with type 1 diabetes and type 2 diabetes, there is alpha-cell dysfunction.

"Patients with diabetes frequently have a deficient GlucoLife Online glucagon response to hypoglycemia and GlucoLife Online exhibit an inappropriately high glucagon response to a meal," explains research from the American Diabetes Association. In layman’s terms: Your body produces too little glucagon when you do need it (like when your blood sugar is dropping) and too much glucagon when you don’t need it (like after eating). The explanation for why this happens is still a bit of a mystery. The alpha-cells are not being attacked and destroyed by your immune system like it does with beta-cells. One proposed theory is that because of the dysfunctional (or non-existant) insulin production, glucagon production isn’t able to accurately determine when it should or shouldn’t increase its production. What can you do about it? I’ve got good news and bad news for you when it comes to glucagon regulation. The good news is that there are a ton of drugs on the market today that reduce the amount of glucose your liver produces - working to counter the pancreas’ overproduction of glucagon.

The bad news is that all of these medications are intended for those with type 2 diabetes, which means getting a hold of them as a person with type 1 means using it "off-label" and likely paying a hefty price for it. This class of drugs helps a person with type 2 diabetes produce more insulin and reduce the amount of glucose produced by the liver. Now, for a person with type 1, there’s no point in trying to produce more insulin because the beta-cells will still be destroyed, but these medications should still impact the production of glucose by the liver. This class of drugs works entirely by reducing the amount of glucose produced by your liver. This inevitably makes you more sensitive to insulin. It also reduces the amount of sugar you absorb from the food you eat and reduces your appetite. And many type 1s have been experimenting lately with Glucophage, and seeing the benefits, particularly after eating. The only trouble is this class of drugs also comes with some unpleasant digestive side-effects. If you can get the "extended-release" version, you’ll likely be more comfortable. At the end of the day, many of us can’t start taking an additional drug we would have to pay for entirely out-of-pocket because they aren’t approved by the FDA for type 1 diabetes. But it’s helpful to understand that you have yet another variable working against you. That doesn’t mean you should give up. It means you shouldn’t be so hard on yourself when things don’t go perfectly.

Starch is a carbohydrate found in foods like bread, pasta, rice, and potatoes, and it quickly raises blood sugar and insulin on Keto. Resistant starch acts more like fiber, but foods containing it still come packaged with regular starch. This guide explains how starch works and the best low-carb alternatives. Going Keto means cutting carbs from your diet. The forbidden carbs fall into two main categories: sugar and starch. This article is about starch. Starch, which is just a cluster of sugar molecules, is found in foods like rice, bread, pasta, potatoes, yams, and carrots. Since the Agricultural Revolution about 12,000 years ago, starch has dominated the human diet. Once we started growing crops, we hung up the hunting spears. But 12,000 years is nothing in evolutionary terms. In the millions of years before agriculture, hominids evolved to flourish without a steady supply of carbs. We evolved, in other words, to cycle in and out of ketosis.

That’s how our ancestors stayed lean and metabolically flexible. Taking a break from starch can help you activate your fat-burning machinery too. Today you’ll learn what starch is, why it’s prohibited on the Keto diet, and how to replace starch with low-carb alternatives. Starch is a type of carbohydrate, the primary way in which plants store energy. Plants form carbs via photosynthesis and store them as starch and sugar. Animals eat plants and store the digested sugar (glucose) as glycogen. Starch is made up of molecules called polysaccharides. In layman's terms, this means that starches are made of chains of sugar molecules. That sugar molecule is called glucose. It’s the same molecule that arrives in your blood as blood sugar (blood glucose) when you digest starch. The two main starch molecules are called amylose and amylopectin. When you eat amylose or amylopectin, an enzyme called amylase breaks them into glucose for easy absorption through the small intestine.